From Quarks to Quasars » The Center of a Black Hole: Infinitely Massive Singularity or Portal into another Universe?
Black holes are one of the most naggingly peculiar objects in the universe. Beyond the event horizon of a black hole, our equations are turned upside down; they also get turned inside out when we attempt to fathom the singularity at its center when using the equations given to us by Einstein. To make life simpler, what if we removed the singularity all together? There is some math for that.
Quantum gravity is an attempt in theoretical physics to explain gravity and the behavior of gravitational fields at the quantum scale. In other words, quantum gravity is one possible ‘Theory of Everything’ scientists are considering. When you apply the framework of quantum gravity to a black hole, some very interesting things happen, among the most interesting is the vanishing singularity.
Instead of a singularity, quantum gravity replaces the center of a black hole with science-fiction’s best friend – a portal to another universe. How many times have we seen our hero (or the villain) fall into a black hole and avoid a crushing death by being transported to another universe? That might not be so far from the truth. Disregarding the fact that our favorite sci-fi movies get a boost of scientific accuracy, such a model immediately helps physicists resolve the black hole information paradox.
The paradox basically addresses two parts of scientific theory that are butting heads with each other. On one hand, general relativity combined with quantum mechanics seems to suggest that information can permanently vanish when it’s devoured by a black hole. In contrast, a common tenet of science states that information cannot be permanently destroyed.
OK, back to the singularity, or lack thereof. As most of you are aware, flying into a black hole is a very poor life choice. According to relativity, tidal forces from the black hole will elongate you in a process affectionately called ‘spaghettification’ – and all of this happens before you cross the event horizon. After you pass the point of no return, you’ll continue to fall to the singularity (the point at the center of the black hole where gravity is infinitely strong and all matter is crushed into an infinitely dense point–fun times). What happens next? We have no idea. General relativity simply stops working and breaks down when trying to describe the singularity.
Singularities aren’t the only thing relativity has problems with. Einstein’s crowning achievement also breaks down when describing the big bang. In 2006, a team of physicists used loop quantum gravity in an attempt to explain the big bang; their results were very interesting. Again, the singularity commonly thought to exist at the start of the universe disappeared and was replaced with something the team described as a “quantum bridge” that brought the team into an older universe that existed before ours.
Relativity is a fascinating theory that is nothing short of remarkable, but maybe it’s playing with an incomplete deck when it comes to black holes and their inner singularities. Perhaps a comprehensive theory of everything will reveal hidden portals within one of nature’s most fearsome creations.
Dec. 17, 2013 — Imagine kicking a cocaine addiction by simply popping a pill that alters the way your brain processes chemical addiction. New research from the University of Pittsburgh suggests that a method of biologically manipulating certain neurocircuits could lead to a pharmacological approach that would weaken post-withdrawal cocaine cravings. The findings have been published in Nature Neuroscience.
Researchers led by Pitt neuroscience professor Yan Dong used rat models to examine the effects of cocaine addiction and withdrawal on nerve cells in the nucleus accumbens, a small region in the brain that is commonly associated with reward, emotion, motivation, and addiction. Specifically, they investigated the roles of synapses -- the structures at the ends of nerve cells that relay signals.
When an individual uses cocaine, some immature synapses are generated, which are called "silent synapses" because they send few signals under normal physiological conditions. After that individual quits using cocaine, these "silent synapses" go through a maturation phase and acquire the ability to send signals. Once they can send signals, the synapses will send craving signals for cocaine if the individual is exposed to cues that previously led him or her to use the drug.
The researchers hypothesized that if they could reverse the maturation of the synapses, the synapses would remain silent, thus rendering them unable to send craving signals. They examined a chemical receptor known as CP-AMPAR that is essential for the maturation of the synapses. In their experiments, the synapses reverted to their silent states when the receptor was removed.
"Reversing the maturation process prevents the intensification process of cocaine craving," said Dong, the study's corresponding author and assistant professor of neuroscience in Pitt's Kenneth P. Dietrich School of Arts and Sciences. "We are now developing strategies to maintain the 'reversal' effects. Our goal is to develop biological and pharmacological strategies to produce long-lasting de-maturation of cocaine-generated silent synapses."
The above story is based on materials provided by University of Pittsburgh. The original article was written by Melissa Carlson.
The researchers hypothesized that if they could reverse the maturation of the synapses, the synapses would remain silent, thus rendering them unable to send craving signals. They examined a chemical receptor known as CP-AMPAR that is essential for the maturation of the synapses. In their experiments, the synapses reverted to their silent states when the receptor was removed.
"Reversing the maturation process prevents the intensification process of cocaine craving," said Dong, the study's corresponding author and assistant professor of neuroscience in Pitt's Kenneth P. Dietrich School of Arts and Sciences. "We are now developing strategies to maintain the 'reversal' effects. Our goal is to develop biological and pharmacological strategies to produce long-lasting de-maturation of cocaine-generated silent synapses."
The above story is based on materials provided by University of Pittsburgh. The original article was written by Melissa Carlson.